Fungal pathogens present a significant threat to global health. As eukaryotes, they share considerable homology with their hosts,
necessitating the development of innovative, non-cross-reactive therapies. Our group combines several approaches to achieve this goal by answering two broad questions: how do fungi overcome immune surveillance and cause infectious diseases and, conversely, how the host immune response can protect against fungal pathogens? or in other words, what makes a microbe a pathogen?